@inproceedings{BicknellTassiusSchollBeckeretal.1992,
  author    = {Bicknell-Tassius, R. N. and Scholl, S. and Becker, Charles R. and Landwehr, G.},
  title     = {High magnetic field transport in II-VI heterostructures},
  url       = {http://nbn-resolving.de/urn:nbn:de:bvb:20-opus-37797},
  year      = {1992},
  abstract  = {In the present work we report the results of magneto-transport measurements on some Hg-based li-VI semiconductor epitaxiallayers grown by molecular beam epitaxy. The transport measurement were carried out at temperatures in the range 0.4 - 4.2 K in magnetic fields up to 10.0 T. Further, we point out the necessity of using multicarrier models for data interpretation and show finally some Shubnikov-de-Haas results on sampies with high mobility carners.},
  language  = {en}
}
@article{GeisGeickBeckeretal.1977,
  author    = {Geis, G. and Geick, R. and Becker, Charles R. and Wagner, V.},
  title     = {Antiferromagnetic resonance in CoO/NiO mixed crystals},
  url       = {http://nbn-resolving.de/urn:nbn:de:bvb:20-opus-31239},
  year      = {1977},
  abstract  = {We have studied the lowest magnetic excitation of Ni\(_{1-x}\)Co\(_x\)O mixed crystals for 0.94 \(\leq\) x \(\leq\) 1. Together with previous results for 0.02 \(\leq\) x \(\leq\) 0.07 and neutron data for x = 0.14 and x = 0.30, the results are discussed by means of a model, especially the variation of AFMR frequency and preferred spin direction with Co concentration x.},
  language  = {en}
}
@article{WuBeckerWaagetal.1991,
  author    = {Wu, Y. S. and Becker, Charles R. and Waag, A. and Bicknell-Tassius, R. N. and Landwehr, G.},
  title     = {The effects of laser illumination and high energy electrons on molecular-beam epitaxial growth of CdTe},
  url       = {http://nbn-resolving.de/urn:nbn:de:bvb:20-opus-30795},
  year      = {1991},
  abstract  = {We report the results of a detailed investigation on the Te-stabilized (2 x 1) and the Cdstabilized c( 2 X 2) surfaces of ( 100) CdTe substrates. The investigation demonstrates for the first time that both laser illumination and, to a greater extent, high-energy electron irradiation increase the Te desorption and reduce the Cd desorption from ( 100) CdTe surfaces. Thus it is possible by choosing the proper growth temperature and photon or electron fluxes to change the surface reconstruction from the normally Te-stabilized to a Cd-stabilized phase.},
  language  = {en}
}
@article{RitterFanPaulsonetal.2016,
  author    = {Ritter, Cathrin and Fan, Kaiji and Paulson, Kelly G. and Nghiem, Paul and Schrama, David and Becker, J{\"u}rgen C.},
  title     = {Reversal of epigenetic silencing of MHC class I chain-related protein A and B improves immune recognition of Merkel cell carcinoma},
  series = {Scientific Reports},
  journal   = {Scientific Reports},
  number    = {21678},
  edition   = {6},
  doi       = {10.1038/srep21678},
  url       = {http://nbn-resolving.de/urn:nbn:de:bvb:20-opus-167992},
  year      = {2016},
  abstract  = {Merkel cell carcinoma (MCC) is a virally associated cancer characterized by its aggressive behavior and strong immunogenicity. Both viral infection and malignant transformation induce expression of MHC class I chain-related protein (MIC) A and B, which signal stress to cells of the immune system via Natural Killer group 2D (NKG2D) resulting in elimination of target cells. However, despite transformation and the continued presence of virally-encoded proteins, MICs are only expressed in a minority of MCC tumors in situ and are completely absent on MCC cell lines in vitro. This lack of MIC expression was due to epigenetic silencing via MIC promoter hypo-acetylation; indeed, MIC expression was re-induced by pharmacological inhibition of histone deacetylases (HDACs) both in vitro and in vivo. This re-induction of MICs rendered MCC cells more sensitive to immune-mediated lysis. Thus, epigenetic silencing of MICs is an important immune escape mechanism of MCCs.},
  language  = {en}
}
@article{ReuterHaufImdahletal.2023,
  author    = {Reuter, Christian and Hauf, Laura and Imdahl, Fabian and Sen, Rituparno and Vafadarnejad, Ehsan and Fey, Philipp and Finger, Tamara and Jones, Nicola G. and Walles, Heike and Barquist, Lars and Saliba, Antoine-Emmanuel and Groeber-Becker, Florian and Engstler, Markus},
  title     = {Vector-borne Trypanosoma brucei parasites develop in artificial human skin and persist as skin tissue forms},
  series = {Nature Communications},
  volume    = {14},
  journal   = {Nature Communications},
  doi       = {10.1038/s41467-023-43437-2},
  url       = {http://nbn-resolving.de/urn:nbn:de:bvb:20-opus-358142},
  year      = {2023},
  abstract  = {Transmission of Trypanosoma brucei by tsetse flies involves the deposition of the cell cycle-arrested metacyclic life cycle stage into mammalian skin at the site of the fly's bite. We introduce an advanced human skin equivalent and use tsetse flies to naturally infect the skin with trypanosomes. We detail the chronological order of the parasites' development in the skin by single-cell RNA sequencing and find a rapid activation of metacyclic trypanosomes and differentiation to proliferative parasites. Here we show that after the establishment of a proliferative population, the parasites enter a reversible quiescent state characterized by slow replication and a strongly reduced metabolism. We term these quiescent trypanosomes skin tissue forms, a parasite population that may play an important role in maintaining the infection over long time periods and in asymptomatic infected individuals.},
  language  = {en}
}
@article{DindasScherzerRoelfsemaetal.2018,
  author    = {Dindas, Julian and Scherzer, S{\"o}nke and Roelfsema, M. Rob G. and Meyer, Katharina von and M{\"u}ller, Heike M. and Al-Rasheid, K. A. S. and Palme, Klaus and Dietrich, Petra and Becker, Dirk and Bennett, Malcolm J. and Hedrich, Rainer},
  title     = {AUX1-mediated root hair auxin influx governs SCFTIR1/AFB-type Ca2+ signaling},
  series = {Nature Communications},
  volume    = {9},
  journal   = {Nature Communications},
  doi       = {10.1038/s41467-018-03582-5},
  url       = {http://nbn-resolving.de/urn:nbn:de:bvb:20-opus-225368},
  year      = {2018},
  abstract  = {Auxin is a key regulator of plant growth and development, but the causal relationship between hormone transport and root responses remains unresolved. Here we describe auxin uptake, together with early steps in signaling, in Arabidopsis root hairs. Using intracellular microelectrodes we show membrane depolarization, in response to IAA in a concentration- and pH-dependent manner. This depolarization is strongly impaired in aux1 mutants, indicating that AUX1 is the major transporter for auxin uptake in root hairs. Local intracellular auxin application triggers Ca2+ signals that propagate as long-distance waves between root cells and modulate their auxin responses. AUX1-mediated IAA transport, as well as IAA- triggered calcium signals, are blocked by treatment with the SCFTIR1/AFB - inhibitor auxinole. Further, they are strongly reduced in the tir1afb2afb3 and the cngc14 mutant. Our study reveals that the AUX1 transporter, the SCFTIR1/AFB receptor and the CNGC14 Ca2+ channel, mediate fast auxin signaling in roots.},
  language  = {en}
}